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Irbesartan stops angiotensin II binding on the AT1 receptor in tissues like vascular easy muscle mass along with the adrenal gland. Irbesartan and its active metabolite bind the AT1 receptor with 8500 situations far more affinity than they bind towards the AT2 receptor.
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Irbesartan blocks the vasoconstrictor and aldosterone-secreting effects of angiotensin II by selectively binding for the AT1 angiotensin II receptor. You can find also an AT2 receptor in many tissues, but It is far from involved with cardiovascular homeostasis.
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Normally, sodium is reabsorbed into epithelial cells in the distal convoluted tubule and pumped into the basolateral interstitium by a sodium-potassium ATPase, making a concentration gradient involving the epithelial cell plus the distal convoluted tubule that encourages the reabsorption of h2o.
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Blockade from the arena plus AT1 receptor gets rid of the damaging feedback of angiotensin II on renin secretion, though the resulting greater plasma renin activity and circulating angiotensin II tend not to conquer the consequences of irbesartan on blood pressure level.
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